Sapna Gupta, Associate Professor
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CH 251, Respiratory Drugs, Skeletal Muscle Relaxants, Antihypertensive Drugs, Angina Pectoris, Cardiac Arrhythmias, Congestive Heart Failure, Coagulation Disorders and Hyperlipidia, Endocrine Pharmacology, Male and Female Hormones, Thyroid and Parathyroid, Diabetes

Final Exam Objectives

 


SKELETAL MUSCLE RELAXANTS

 Spasticity: exaggerated muscle stretch reflux caused by loss of control in spinal cord or brain.  Caused by cerebral accident, traumatic lesions in the brain or multiple sclerosis.

Spasms: increased tension, usually involuntary

Two kinds of muscle relaxants:  Centrally acting (act on spinal cord) and direct acting (on the muscle) 

Treatment

Used in

Mechanism

Adverse effects

Centrally Acting

 

 

 

Baclofen

Spasticity associated with lesions of spinal cord.  Good in MS.

Given orally and intrathecally in severe cases.

Releases GABA

Drowsiness, confusion, hallucinations. Adverse effects are individual based.

Diazepam (Valium)

Patients with cord lesions and cerebral palsy

 

Sedation, tolerance and dependance.

Combination

Other drug mechanisms are not understood well and are generally used as adjunct to PT.  Most drugs are combined with analgesics

Eg Parafon Forte (clorozoxazone and acetaminophen)

Norgesic (orphenadrine and aspirin)

Dizziness, headaches

Direct Acting

 

 

 

Dantrolene Sodium (Dantrium)

Traumatic cord lesions, advanced MS, cerebral palsy.

(not used for muscle spasms)

Inhibits release of calcium within muscle

Muscle weakness, severe hepatotoxicity

Other eg Botulinum toxin (Botox)

 

Prevents release of acetylcholine

 

 Spasms:  can be treated by opioid or non-opioid analgesics.  Most common side effect is sedation.



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