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Using half a Deca-unit and half a Dianabol-unit per week (say 200 mg/week Deca and 20 mg/day Dianabol) gives better gains than using one unit of either alone. npc bodybuilding, injectable steroids, wholesale bodybuilding supplements, steroids books, anabolic steroid cycle, female muscle worship, steroid powder, anabolic steroids for sale in the uk, women steroids, steroids pictures Illegal anabolic steroids. This effect is called synergy and results when there is more than one mechanism of action. The above math remains correct for any given receptor but this is saying that there are more things going on in the body than simply binding to one receptor. Aside from this and other practical but well-confirmed observations, there is scientific evidence that this is indeed the case. npc bodybuilding, injectable steroids, wholesale bodybuilding supplements, steroids books, anabolic steroid cycle, female muscle worship, steroid powder, anabolic steroids for sale in the uk, women steroids, steroids pictures Female muscle worship. Scientific Evidence for Multiple Modes of ActionThe first thing to consider is whether or not a single mode of action is sufficient to explain all results, as with the simplest case described for Drugs A and B, or whether data is in conflict with such a model. The equation given earlier allows one, given a measured Kd value, to calculate what percentage of receptors is occupied for a given concentration of drug. The Kd value for testosterone and the androgen receptor (AR) actually is not known with great precision for humans, but is approximately . npc bodybuilding, injectable steroids, wholesale bodybuilding supplements, steroids books, anabolic steroid cycle, female muscle worship, steroid powder, anabolic steroids for sale in the uk, women steroids, steroids pictures Steroids pictures. 44 nmol/L. 1 Free testosterone levels in normal men average approximately . 07 nmol/L. 2,3,4Contrary to previous statements made by me (although those statements had been made in the scientific literature) this indicates that normal testosterone levels are not sufficient to saturate the AR. The equation given shows that with these values for free testosterone (Tf) and for Kd, one would expect only 14% of ARs to be occupied at any time. Increasing Tf by ten times would improve this to 61% occupancy, which still is not saturated. Increasing twenty times would yield further improvement to 76%. Perhaps this correlates well with the observation that gains improve markedly relative to low dose as one increases amount of testosterone used to 1 gram per week, but going to 2 grams per week offers only a modest further increase. These results surprise me and are definitely contrary to accepted wisdom. I can only speculate at the moment that those who were trying to determine whether or not receptors are saturated made the mistake of performing the calculation with total testosterone levels instead of Tf. Doing so would lead to that conclusion but is an incorrect method. I had been going to argue as I had previously that the dose response curve, which extends at least to the 1 gram per week level,5 indicates that there must be more than one mechanism of action, since response increases even past the point of saturation. However these calculations just performed indicate that the dose response curve, through the range that has been studied, is in accord with known values for Kd. This doesn't prove that there is only one mechanism, but just that one mechanism is not disproven by the dose response curve. Is there other evidence for multiple mechanisms?Yes. First, there are indisputably molecular targets that are not ARs within some cells which bind androgen and give pharmacological response to androgen. These targets may well have (and in some cases are shown to have) quite different binding properties than the AR does. One AAS might be more potent than another at the AR, but less potent at this other target. Now these targets are not well known or characterized at all, but there is compelling evidence for their existence. First, as discussed above, for any given target (or receptor) drugs acting only at that receptor will behave the same way and differ only in their potencies. Now if all AAS behaved the same way and differed only in their potencies, and had the same ratios of potency regardless of the activity being studied (whether in muscle or skin or nerves, etc. ) then this would be consistent with there being only one target or receptor. However, if some AAS are effective in some activities but do nothing in others, while other AAS do have these other activities, then this can't all be occurring from the same receptor. Most of the research in this area is rather far removed from bodybuilding, but the principles still apply. Biochemistry is usually much broader than any one specific cell being studied. (For example, most human biochemistry was actually learned originally by study of E.
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