Parts and Pieces Your
normal mouse has a long straight tail, medium to large ears, and all the
other “normal” parts and pieces. Some mice are missing some of their parts
and pieces, and/or they have strange convolutions of those same parts. This
is a very short section, but in truth there are a lot of genes that cause
mice to change their parts and pieces, anywhere from multiple toes on their
feet, to hammertoes, curly tails, fat tails, funny ears, etcetera. To find
more of these genes a very fascinating resource is the Jackson Labs website. |
||
Symbol |
Photo |
Description |
Chr. 9 |
Short Ears Homozygotes have short, slightly ruffled external ears recognizable at about 14 days of age. These mice have other abnormalities of the spine, including fewer ribs, missing wrist and ankle bones, and other problems. |
|
Grem Chr. Un |
Gremlin. A dominant gene with only
limited penetrance. This mouse is notable for having one ear located on the
side of its head while the other ear is located in the normal position.
|
|
GremD Chr. Un |
Dumbo. Dumbo
mice are an extension of the gremlin gene. Dumbo mice have two lowered ears
usually recognizable by two to three weeks of age. |
|
Chr. Un |
Porcine Tail. Tail varies from a normal tail to a tightly looped tail as in a pig.
Some of these mice show waltzing behavior. |
|
Chr. 17 |
Tailless.
The dominant gene T results in tailless mice T/~ are tailless.
There are modifiers to the t gene which have several results: tn/~
mice have normal tails. T/tn mice are also tailless, and T/T
and tn/tn mice are homozygous lethal. Apparantly the t-type sperm has a tendency
to fertilize more ova then non-t sperm, leading to a higher proportion of
tailless mice than would be expected by mendelian genetics. This is perhaps
responsible for the high incidence in the wild, despite its being homozygous
lethal. |
[Mendelian Genetics | Beyond Mendel]
[The Five Basics | The Markings | Coat Consistency]
[Parts and Pieces | Genetic Mysteries]