Pharmacokinetics, Pharmacodynamics, Pharmacology of Autonomic nervous system drugs, Autocoids

 

 

 

1.    The apparent volume of distribution of a drug.

 

a.     T: basic drugs are more widely bound to tissue proteins.

b.     F: Vd relates the amount of drug in the body to its TOTAL concentration in the plasma.

c.     F

d.     T: in cardiac failure, cardiac output decreases which in turn reduces drug distribution to the peripheral sites.

 

 

2.   Binding of drugs to plasma proteins show the following characteristics:

 

a.     F: only the free unbound drug fraction is pharmacologically active as it can pass through the capillary membrane to various tissues.

b.     F: the tendency to cause adverse effects is dependent on the therapeutic index of the drug and the amount displaced.

c.     F: some acidic drugs like aspirin are strongly bound to plasma proteins but not to tissues.

d.     T: may be due to accumulation of some organic molecules which can displace a drug from albumin binding sites.

 

 

3.    Drugs administered by the oral route show the following characteristics:

 

a.     T

b.     T

c.     F: the small intestine is the major site of absorption for all drugs due to its much larger surface area for absorption and the high rate of intestinal blood flow.

d.     F: generally, a basic drug is more soluble in acid pH than in alkaline pH.

 

 

4.    The apparent volume of distribution of a drug:

 

a.     F: it is a useful proportionality constant which indicates the relative distribution of a drug between plasma and its potential binding sites.

b.     T 

c.     T: the longer the half-life, the greater the amount of drug present in the body.

d.     T

 

 

5.    Acetylcholine:

 

a.      T

b.      T

c.      T

d.      T: binds to M2 muscarinic receptors which couple with Gi and Gk leading to activation of potassium channels with consequent hyperpolarization of cardiac myocytes.

 

 

6.    D-tubocurarine:

 

a.      F: is a quarternary amine, therefore poorly absorbed.

b.      T: at higher doses, cause blockade by occlusion of open ion channels.

c.      T: increase acetylcholine concentration at junctional cleft to displace D-tubocurarine from the nicotinic sites.

d.      T

 

 

7.    Propranolol:

 

a.     F: is a pure antagonist.

b.     F: is a non-specific beta blocker.

c.     T: due to blockade of beta2 blockers in bronchial smooth muscle.

d.     T

 

 

8.    Histamine causes:

 

a.     T

b.     T

c.     T

d.     T

 

 

9.    The following are physiological antagonists of histamine acting on bronchial smooth muscle:

 

a.      T

b.      T

c.      F

d.      F

 

 

10.    Antihistamines antagonize histamine action by:

 

a.      F

b.      T

c.      F

d.      F

 

 

 

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