Mucoid Pseudomonas in Cystic Fibrosis

Infection with P. aeruginosa begins in childhood, even in the early months of life, with nonmucoid strains being acquired first.

1.       What is a “mucoid” strain of pseudomonas aeruginosa?

·            A mutant strain of pseudomonas aerug. that produces an alginate coat that give them a mucoid appearance

·            Thought to be in response to neutrophils that attempt to kill the bacteria via peroxides

Mucoid conversion of Pseudomonas aeruginosa by hydrogen peroxide: a mechanism for virulence activation in the cystic fibrosis lung.

Mathee K; Ciofu O; Sternberg C; Lindum PW; Campbell JI; Jensen P; Johnsen AH; Givskov M; Ohman DE; Molin S; Hoiby N; Kharazmi A

Microbiology 1999 Jun;145 ( Pt 6):1349-57

To mimic the inflammatory environment of the CF lung, P. aeruginosa PAO1, a typical non-mucoid strain, was grown in a biofilm. This was treated with low levels of H2O2, as if released by the PMNs, and the formation of mucoid variants was observed.

When a P. aeruginosa biofilm was repeatedly exposed to activated PMNs in vitro, mucoid variants were also observed, mimicking in vivo observations. Thus, PMNs and their oxygen by-products may cause P. aeruginosa to undergo the typical adaptation to the intractable mu- coid form in the CF lung.

 

2.       What is the significance of the alginate (polysaccharide) coat?

·          Alginate that is produced by mucoid Pseudomonas aeruginosa interferes with phagocytic killing
 

·          Immune system is unable to kill these mucoid organisms, promoting pseudomonas aerug. colonization of the lungs

·         
 
This mucoid phenotype is seen infrequently in non-CF populations, but occurs in over 75 percent of P. aeruginosa isolated from patients with CF

 

3.       What is the significance of pseudomonas aerug. colonization of the lungs?

·        Patients with CF die from respiratory failure because of a gradual loss of lung function. This results from the chronic inflammatory process perpetuated by P. aeruginosa, which colonizes the lung for periods of up to 40 years. The inflammation is triggered by the host response to the organism, not by tissue invasion.

·        Epidemiologic studies show that chronic infection with Pseudomonas aeruginosa is an independent risk factor for accelerated loss of pulmonary function and decreased survival.

 

4.       Are these mucoid strains resistant to anti-biotics?  Not based on alginate coat

These organisms are extremely difficult to eradicate once established, and can be isolated repeatedly over the course of many years. This phenomenon is due to:
 

·          Poor penetration of the antibiotics into purulent airway secretions

·          The alginate that is produced by mucoid Pseudomonas aerug interferes with phagocytic killing

·          Native or acquired antibiotic resistance

·          CF-related defects in mucosal defenses

 

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