Physical effects of steroids
The possibility at least exists. physical effects of steroids Sports steroids. Speaking of ductal branching morphogenisis in the developing rat prostate,6 here indeed different steroids behave differently. While to the AR testosterone is less potent than DHT, here the reverse relationship was found. Furthermore, methyltrienolone, which is a more potent agonist (activator) of the AR than is DHT, was no more effective than DHT in inducing ductal branching and was less effective than testosterone. physical effects of steroids Anabolic video productions. This cannot be explained by assuming that aromatization of testosterone to estradiol contributed to the process, because 5a -androstan-3a ,17b -diol (which cannot aromatize) was similarly potent. Thus, there is some target or receptor in these tissues which has different "preferences" (Kd values, and different rank order of potency) than the AR does. Could this also be the case for muscle growth? Perhaps. physical effects of steroids Effect of anabolic steroids. Another example is found in the virilization of the mammary gland of female rats. 7 The same results are seen here as in the above example of the rat prostate. Testosterone (T) has more activity than DHT does, though at the AR that would not be so. Differences also are seen in the male accessory glands of the rabbit and rat. 8 Testosterone propionate and DHT propionate were found to be equally potent in supporting growth and secretory activity of these glands, but the above-mentioned 5a -androstan-3a ,17b -diol was considerably more potent than these in the prostate but completely ineffective in the epidydimis. Furthermore, use of an antiandrogen (AR blocker) did not affect the function of the epidydimis at all. Thus, the activity of testosterone and DHT in this tissue is not via the AR. Are there muscle-building activities that are not via the AR? If the mechanism exists in one tissue it probably does in others as well. Here is an activity that is itself of more interest: regulation of lipolysis (fat release) in adipocytes (fat cells). 9 T, but not DHT, stimulated catecholamine-induced lipolysis. The findings indicated that T but not DHT induced upregulation of b -adrenergic receptors. Use of an aromatase inhibitor did not change these results, so conversion to estrogen was not responsible for the difference. If this activity were via the AR, DHT would also have exhibited this effect. Clearly then, something is going on that is not via the AR. Differential effects of different AAS on human fat cells have also been seen. 10 Oxandrolone was most effective in reducing subcutaneous abdominal fat and visceral fat in obese middle- aged men while weight did not change, as a result of muscle mass increase. Testosterone enanthate gave a small decrease in subcutaneous fat but a slight increase in visceral fat. Nandrolone decanoate also increased visceral fat while decreasing subcutaneous fat. If these activities were via the AR, all three steroids should give the same effects, differing only in potency or the dosage required. There are some interesting studies on sexual receptivity of female rats. Methyltestosterone, methandrostenolone (Dianabol), nandrolone decanoate, and stanozolol all interfered with sexual receptivity (a different result than seen in human bodybuilders) while testosterone propionate did not. 11In male rats,12,13,14 differential activities are also seen.
Physical effects of steroids
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