NMS

6/7/99

• Parasympathetic atonia—loss of parasympathetic output, neurological lesion, never normal, disease

• Parasympatheticotonia—increase in parasympathetic (nausea, vomiting), normal reflex, expected when needed, if occurs constantly this is dis-ease
• ex: vagotonia (Vagus is largest parasympathetic nerve)
• #1 cause of bradycardia (<60bpm)
• AV block (excessive vagal tone)
• Adam Stokes—atropine—vagal inhibitor, block affects of vagal nerve
• Allopathic model—meds to create a neurological lesion
• Sympathetics are at a normal level (chiropractic) would you want to bring sympathetics up to a higher level to balance
• What could cause the Vagus to increase
• Ch 12, area of spine has a direct impact on Vagus (upper cervical area)
• Upper cervical area adds to vagal tone
• Hypertonic muscles around upper cervical area
• Fixated
• Tender
• Viscerosomatic reflex (osteopathic)—subluxation
• Somatoviceral reflex (chiropractic)
• Old chiropractic would say decreased sympathetics (bone on nerve)

• Restore homeostasis
• In hypertonia, the sympathetics are elevated trying to restore homeostasis but the parasympathetics are so far out of control that the sympathetics can’t catch up