NMS
6/7/99
- Parasympathetic atonia—loss of parasympathetic output, neurological lesion, never normal, disease
- Parasympatheticotonia—increase in parasympathetic (nausea, vomiting), normal reflex, expected when needed, if occurs constantly this is dis-ease
- ex: vagotonia (Vagus is largest parasympathetic nerve)
- #1 cause of bradycardia (<60bpm)
- AV block (excessive vagal tone)
- Adam Stokes—atropine—vagal inhibitor, block affects of vagal nerve
- Allopathic model—meds to create a neurological lesion
- Sympathetics are at a normal level (chiropractic) would you want to bring sympathetics up to a higher level to balance
- What could cause the Vagus to increase
- Ch 12, area of spine has a direct impact on Vagus (upper cervical area)
- Upper cervical area adds to vagal tone
- Hypertonic muscles around upper cervical area
- Fixated
- Tender
- Viscerosomatic reflex (osteopathic)—subluxation
- Somatoviceral reflex (chiropractic)
- Old chiropractic would say decreased sympathetics (bone on nerve)
- Restore homeostasis
- In hypertonia, the sympathetics are elevated trying to restore homeostasis but the parasympathetics are so far out of control that the sympathetics can’t catch up