NMS
6/10/99
Hello every one J
-Tom
Good Bye L
-Tom
- Dyspnea—shortness of breath
- Most people believe that if you are short of breath have decreased O2
- If really have decreased O2, become lightheaded, tired, memory failure
- Dyspnea is a cortical function, has no relationship to blood gas
- It is a proprioceptive disorder
- Proprioceptors in ribs, intercostal mm, facets (when they are moving)
- Person w/no pulmonary problems, bind their ribs and let them sit, they would experience short of breath but blood gases would be fine
- Mechanoreceptor deep in the lung tissue that senses shortness of breath—J receptor (juxtaalveolar—next to alveolar)—when stimulated send message to brain of dyspnea
- NE is released into pulmonary tissue
- An adjust will have long and short term affects
- Short—normal motion to ribs and costal joints
- Long—reduce the amount of NE in tissues, which will reduce the output from the J receptors
Muscle Spasm
- CES—central excitatory state
- Dorsal horn—sensory
- Lateral horn—sympathetics
- Ventral horn—motor
- Afferntation—increased sensitivity—sprain/strain—ligament, muscle, fascia
- Goes into the dorsal horn and released neurotransmitters
- Goes to interneurons
- Lateral spinothalamic tract to brain
- May have hurt ligament, muscle or fascia in the neck but are complaining of pain in headaches, shoulder, mid back, interscapular region
- Does not dermatome pain
- Sclerotome is the type of pain
- The interneuron can go and stimulate the lateral horn increasing the sympathetics
- Can go to stimulate the anterior horn leading to spasm
- Frozen shoulder sydromes go along w/whiplash injuries
- ROM, cervical spine adjustments, shoulder adjustmesnt
- Reduces symptoms to bring down CES to homeostatis