NMS

6/10/99

Hello every one J

-Tom

Good Bye L

-Tom

• Dyspnea—shortness of breath
• Most people believe that if you are short of breath have decreased O2
• If really have decreased O2, become lightheaded, tired, memory failure
• Dyspnea is a cortical function, has no relationship to blood gas
• It is a proprioceptive disorder
• Proprioceptors in ribs, intercostal mm, facets (when they are moving)
• Person w/no pulmonary problems, bind their ribs and let them sit, they would experience short of breath but blood gases would be fine
• Mechanoreceptor deep in the lung tissue that senses shortness of breath—J receptor (juxtaalveolar—next to alveolar)—when stimulated send message to brain of dyspnea
• NE is released into pulmonary tissue
• An adjust will have long and short term affects
• Short—normal motion to ribs and costal joints
• Long—reduce the amount of NE in tissues, which will reduce the output from the J receptors

Muscle Spasm

• CES—central excitatory state
• Dorsal horn—sensory
• Lateral horn—sympathetics
• Ventral horn—motor
• Afferntation—increased sensitivity—sprain/strain—ligament, muscle, fascia
• Goes into the dorsal horn and released neurotransmitters
• Goes to interneurons
• Lateral spinothalamic tract to brain
• May have hurt ligament, muscle or fascia in the neck but are complaining of pain in headaches, shoulder, mid back, interscapular region
• Does not dermatome pain
• Sclerotome is the type of pain
• The interneuron can go and stimulate the lateral horn increasing the sympathetics
• Can go to stimulate the anterior horn leading to spasm
• Frozen shoulder sydromes go along w/whiplash injuries
• ROM, cervical spine adjustments, shoulder adjustmesnt
• Reduces symptoms to bring down CES to homeostatis